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Our study suggests that inhibition of fibrin deposition in severe asthma may be a therapeutic approach. 我們研究表明，抑制嚴(yán)重哮喘患者氣道纖維蛋白沉積可能成為哮喘的治療措施之一。
Objective To investigate the effect of fibrin deposition on endothelial cell function in pulmonary tissue of rats with different ages. 目的探討纖維蛋白沉積對(duì)不同鼠齡大鼠肺組織血管內(nèi)皮細(xì)胞功能的影響。
After adjusted by the value of fibrin deposition, the expressions of ICAM-1 and PAI-1 in aged rats were significantly higher than those in young rats(P0.05). 經(jīng)纖維蛋白沉積量校正后，老年大鼠肺組織ICAM-1和PAI-1的表達(dá)量仍顯著高于青年大鼠（P0.05）。